Snyder Research
Contact
Position:
Associate Professor
233 South 10th Street
Suite 730
Philadelphia, PA 19107
Contact Number(s):
Publications
Publications
- Multiple and Consecutive Genome Editing Using i-GONAD and Breeding Enrichment Facilitates the Production of Genetically Modified Mice
- Mechanisms of antiviral cytotoxic cd4 t cell differentiation
- Inhibitory Molecules PD-1, CD73 and CD39 Are Expressed by CD8+ T Cells in a Tissue-Dependent Manner and Can Inhibit T Cell Responses to Stimulation
- Hematopoietic cell-mediated dissemination of murine cytomegalovirus is regulated by NK cells and immune evasion
- STING sensing of murine cytomegalovirus alters the tumor microenvironment to promote antitumor immunity
- Advances in cytomegalovirus (CMV) biology and its relationship to health, diseases, and aging
- α2β1 Integrin Is Required for Optimal NK Cell Proliferation during Viral Infection but Not for Acquisition of Effector Functions or NK Cell-Mediated Virus Control
- Stochastic expansions maintain the clonal stability of CD8+ T cell populations undergoing memory inflation driven by murine cytomegalovirus
- Correction: Cross-presentation of a spread-defective MCMV is sufficient to prime the majority of virus-specific CD8+ T cells (PLoS ONE (2010) 5:3 (e9681) DOI: 10.1371/journal.pone.0009681)
- Murine cytomegalovirus infection of melanoma lesions delays tumor growth by recruiting and repolarizing monocytic phagocytes in the tumor
- From vaccine vector to oncomodulation: Understanding the complex interplay between CMV and cancer
- Persistent viral replication and the development of T-cell responses after intranasal infection by MCMV
- Cigarette smoke induces metabolic reprogramming of the tumor stroma in head and neck squamous cell carcinoma
- Polymicrobial sepsis influences NK-cell-mediated immunity by diminishing NK-cell-intrinsic receptor-mediated effector responses to viral ligands or infections
- Monocarboxylate transporter 4 (MCT4) knockout mice have attenuated 4NQO induced carcinogenesis; A role for MCT4 in driving oral squamous cell cancer
- The chemokine receptor CXCR3 promotes CD8+ t cell accumulation in uninfected salivary glands but is not necessary after murine cytomegalovirus infection
- Intratumoral infection by CMV may change the tumor environment by directly interacting with tumor-associated macrophages to promote cancer immunity
- Virus-specific CD8+ T cells infiltrate melanoma lesions and retain function independently of PD-1 expression
- Blocking virus replication during acute murine cytomegalovirus infection paradoxically prolongs antigen presentation and increases the CD8+ T cell response by preventing type I IFN-dependent depletion of dendritic cells
- CMV-specific CD8 T cell differentiation and localization: Implications for adoptive therapies